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In nerves exposed to one hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even in the presence of normoglycemic glucose additional power substrate is required when the tissue vitality demand will increase. Such data are vital as they show that glycogen has an essential function below physiological circumstances. The truth that glycogen supported CAP conduction within the absence of exogenously utilized glucose probably has no worth in ascribing a role to glycogen, since aglycaemia is an artificial experimental procedure, [Healthy Flow Blood](http://gitlab.dstsoft.net/vincekerferd39) and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist prior to 1922 (Frier et al., 2014), thus cannot be glycogen’s principal role. However supplying lactate to axons when tissue power demand increases implies a novel position for brain glycogen and suggests it acts as an power buffer somewhat than an power reserve, whose role is not to single-handedly support conduction, however to act in concert with ambient levels of normoglycemic glucose.
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